Full Description
Environmental Causes and Prevention Measures for Alzheimer's Disease examines the increased incidence of the disease in developed countries and aims to educate neuroscientists, medical practitioners and other educated individuals on new insights into environmental causation, primarily metals. This book looks into the web of evidence around the hypothesis of copper toxicity and the additional role that a high fat diet plays in disease progression and cognition loss. The data and its implications are discussed, along with potential prevention measures. This book will generate excitement and interest among neuroscientists, medical practitioners and other biomedical researchers.
Contents
1. Introduction2. A Little Background on Dementia and Alzheimer's Disease3. Interesting and Important Historical and Demographic Facts About the Epidemic of Alzheimer's Disease Pointing to Environmental Intoxicants Causing the Epidemic4. Candidate Environmental Factors for the Alzheimer's Epidemic Part 1: The Metals - Aluminum, Lead, Mercury, Zinc, Iron, and Copper5. Candidate Environmental Factors for the Alzheimer's Epidemic Part 2: Diet and Other Life Style Factors6. Identification of Copper-2, and Copper in General as Major Environmental Intoxicants in the Alzheimer's Epidemic: The Copper Hypothesis7. Background on Copper, Including Why Copper-2 is so Specifically Neurotoxic8. Inorganic Copper, or Copper-2, Ingestion as a Major Causal Factor for Alzheimer's Disease - The Web of Evidence9. Increased Copper Absorption Resulting from Dietary Changes in Developed Countries as Another Causal Factor in the Alzheimer's Disease Epidemic10. The Copper Hypothesis Fits Nicely with Known Risk Factors and Theories of Alzheimer's Disease Causation11. Prevention Measures Action Items: Two Simple Steps to Eliminate Ingestion of Copper-2, and Dietary Changes to Reduce Copper Absorption12. Failures: What the Government Hasn't Done to Ensure Healthy Drinking Water and Nontoxic Multimineral/Multivitamin Pills13. Treatment of Alzheimer's Disease14. Summary and Conclusions
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